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I just ate an entire medium pepperoni pizza for dinner and my roommate started lecturing me on how I'm gonna die in my 30s.

Does eating fatty and sugary foods make your life any shorter?

  • 1
    +1 for eating an entire medium pizza. It makes you fat, that much I know, I believe that(obesity) can be correlated to earlier death. – picakhu Apr 13 '11 at 6:12
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    Can you quantify what you mean by eating fatty and sugary foods? Do you mean eating nothing but fatty/sugary food, or do you mean occasionaly eating fatty and/or sugary food as well as a range of other foods? – Ardesco Apr 13 '11 at 9:33
  • I love salad and I eat from the deli a lot but when I'm not eating those my other choices are pizza and burgers. I also drink a lot of pop. As for being obese, I doubt that is ever going to happen as I eat very little an I am on the lower end of Gavin a normal weight (close to being underweight). I eat fatty foods in an attempt to increase my weight a bit. – Leo Jweda Apr 13 '11 at 14:54
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    Maybe you should see the film “Supersize me”. The actor ate at fast food restaurants during one month and started to have serious health problems. – Benoit Apr 14 '11 at 7:10
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    @LeoJweda: I have good news for you. As you get older, your concerns about being underweight are likely to vanish completely... – Oddthinking Mar 27 '12 at 15:03
5

To answer your questions:

Soda pops

Sugar-sweetened beverage consumption is associated with a significantly elevated risk of type 2 diabetes, whereas the association between artificially sweetened beverages and type 2 diabetes was largely explained by health status, pre-enrollment weight change, dieting, and body mass index.

Sugar-sweetened and artificially sweetened beverage consumption and risk of type 2 diabetes in men, by Lawrence de Koning, Vasanti S Malik, Eric B Rimm, Walter C Willett, and Frank B Hu

Sugary foods and HCFS

Rates of overweight and obesity have been on a steady rise for decades, and the problems society faces from this and associated metabolic diseases are many. As a result, the need to understand the contributing factors is great. A very compelling case can be made that excess sugar consumption has played a significant role. In addition, fructose, as a component of the vast majority of caloric sweeteners, is seen to be particularly insidious. Evidence shows that fructose bypasses many of the body's satiating signals, thus potentially promoting overconsumption of energy, weight gain, and the development on insulin resistance. It has also been shown to increase uric acid levels, which in turn promotes many of the abnormalities seen in the metabolic syndrome including hypertriglyceridemia. However, the main source of fructose in the diet is high-fructose corn syrup (HFCS), an artificially manufactured disaccharide that is only 55% fructose. This review highlights the fact that limited data are available about the metabolic effects of HFCS compared with other caloric sweeteners. The data suggest that HFCS yields similar metabolic responses to other caloric sweeteners such as sucrose.

The Effect of High-Fructose Corn Syrup Consumption on Triglycerides and Uric Acid, by Theodore J. Angelopoulos, Joshua Lowndes, Linda Zukley, Kathleen J. Melanson, Von Nguyen, Anik Huffman, and James M. Rippe

Fatty foods

Different type of fats have different effect, often, but not always, negative. These are the USDA 2010 "Dietary Guidelines for Americans":

Consume less than 10 percent of calories from saturated fatty acids by replacing them with monounsaturated and polyunsaturated fatty acids.

Consume less than 300 mg per day of dietary cholesterol.

Keep trans fatty acid consumption as low as possible, especially by limiting foods that contain synthetic sources of trans fats, such as partially hydrogenated oils, and by limiting other solid fats.

Dietary Guidelines for Americans

The document has a large section dedicated to the effects of fats and references.

0

A recent study of individuals of Ashkenazi Jewish descent aged 95 and older compared against a database of information of those who did not live as long showed evidence that genetics may play a more significant role in longevity than lifestyle choices such as diet, smoking and drinking alcohol.

According to a recent news article on the study:

The researchers concluded that these findings suggest genetics play a role in lifespan and found that there was no difference in lifestyle factors, including BMI (an indicator of obesity), smoking and alcohol consumption, between the [longer vs shorter lifespan] groups.

The researchers were quick to point out that this isn't a license to behave in an unhealthy manner and it might be argued that 'quality of life' issues become a much bigger factor than longevity at these advanced ages. For example, a high BMI indicating obesity also puts you at risk for type 2 diabetes, which if improperly managed can lead to amputations, loss of vision and have other significant quality of life impacts.

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    This doesn't really answer the question. I looked at the abstract of the original study. They didn't find any difference between one subset of the population (Ashkenazi Jews) who lived exceptionally long (95+) time, and the general population born at the same time. That is interesting, but doesn't tell us whether people with similar genetic backgrounds have different lifespans depending on diet. – Oddthinking Mar 27 '12 at 15:12

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